pagine dedicate a tutta la storia, con foto e video e le leggende sul gatto Norvegese HISTORY AND LEGEND CHARACTER WHITE NORWEGIANS FALSE MYTHS LEGEND STANDARD VIDEO STORY COLORS

Costo adozione di un gattino/cucciolo di gatto Norvegese delle foreste. Tutto quello che devi conoscere prima di acquistare. Gatti hosting gratuito e molto altro... INFORMATION How much does a Norwegian Forest cat kitten cost? How much does a Norwegian cat cost, young or adult? _Cc781905-5cde-3194-bb3b-136bad5cf58d_ It is true, for those approaching the world of purebred cat breeding, the price is one of the important information, which is first asked, but it is also true that it is one of the questions that the more it hurts the sentiment of the kittens breeder .... You may be wondering why some breeder to this question has not answered you or perhaps answered you in an annoyed way ... I would like to introduce you to the merits, explaining why: The reimbursement of expenses that is requested is not enough to cover all the expenses incurred during the year ... it is not easy to explain in 4 words to those who have never known the "behind" the wings ", how many problems and expenses must be incurred in order to have healthy, happy and well socialized cats and kittens. The economic side, as the first parameter to evaluate the adoption, in the eyes of those who may not have slept for a week in a row to assist a mother with problems and perhaps have left 1000 euros at the vet for cesarean delivery, is really demeaning ... Although the person has all the justified reasons to ask for it, often one feels disheartened and without any appreciation for what is being done ... For this I ask you, try to identify yourself for a moment; try to think of the affection that binds us to a kitten that we have seen born and we have grown for 3 months and then having to entrust it to others who approach us hard-nosed writing only "PRICE ???" How would you feel? Try to put yourself in our shoes where you have NOT spared us in any way, to raise them, starting from the probiotic supplement at 27 euros per tube, which is not enough even for 2 days on an average litter ... from the cost of food of the best quality (in my case meat from the supermarket prepared by me) to the awake nights to breastfeed them, to look after them as like children in the stages of growth, they can get sick and even die ..._ cc781905-5cde -3194-bb3b-136bad5cf58d_ e while this happens you have the old man of the house with whom you are facing chemotherapy, with a heart broken by pain, hoping he will make it_cc781905-5cde-3194-3194 -136bad5cf58d_ ... e this is just a small slice of reality ..._ cc781905-5cde-3194-bb3b-136bad5cf58d_ Try to think that on this side of the screen ... there are people with all their emotional baggage and the refund asked is vital for their cats, try to use a kind way ... and please! Do not ask us for discounts ... we are not at the market to buy a t-shirt! -_- ' When it comes to living beings the MOST important thing is the health of the puppy and its parents. Here I will try to briefly explain as much as I can, how I grow and what guarantees my puppy has. My philosophy is that of respecting the nature of the Norwegian cat, trying to give them a life as rich as possible of experiences and close (as far as possible) to freedom - feeding them with natural food -biologically appropriate- followed by a veterinarian nutritionist. Our puppies (but also the parents) are fed with Natural food , they do not use kibble and industrial feed rich in cereals that in the diet of a feline, as a narrow carnivore, should not exist. When your Norwegian puppy leaves my home, he will bring with him his natural diet that if you want, you can easily follow thanks to a simple guide with videos and tutorials which I will provide, otherwise I will recommend some of the industrial brands that are less harmful to our beloved Norwegian cats. When the puppy leaves my house he is able to eat both natural and industrial food, as in the last weeks he stays with his mother and siblings, I insert a part of "Grain free" wet and dry feed, in so that you can have a puppy with a really varied and accurate weaning, that will have no problems di "addiction to a specific brand". The parents of the puppies live at home with me but also outdoors in large "protected" enclosures where they can, play and live in the open air and this protects them from many typical overcrowding diseases. -3194-bb3b-136bad5cf58d_ The sun, the air, being able to hunt, play and socialize in an adequate way, guarantees that they can satisfy their ethological needs, this keeps them happy, balanced cats able to transmit this character to their offspring. I pay particular attention to the socialization of kittens, who grow up at home with me, pampered and spoiled. They are soon put in contact with feline uncles and grandparents, but also humans, in order to develop an even more balanced character . _cc781905-5cde-3194-bb3b- 136bad5cf58d_ An important factor that significantly affects the price of the Norwegian cat kitten, are the costs incurred between veterinarians, genetic laboratories, cardiologists to ascertain that the two parents are not they are carriers of the same pathology, and therefore by mating they could fissarne and genetic characters, with the consequent birth of carriers or even sick puppies. Some general information on the parents of the litters: Parents are tested in 2020 for: fiv, felv (ELISA) -giardia (PCR) -NEGATIVE 2021 Parents tested (genetic): GSD4, PKDef -NEGATIVE- Screening Echocardium for Cardiomyopathy (HCM) and Ultrasound x Renal Polycystosis (PKD) - NEGATIVE 2021 These tests carried out on parents, some are annual while some are genetic so it is enough to have them done only once. They are visible in a folder (on Dropbox) online, of which you will have the link if you are interested in a kitten. More information on these pathologies you can read them here Therefore, when the puppy leaves his home he will have: -Pedigree ANFI -Agreement between the parties with Health Guarantees. - Certificate of good health issued by our veterinarian, 2 Trivalent Vaccines- or Antibody Titration (to be chosen by the new owner) 2 Negative stool tests for parasites + giardia, timbred in the health booklet by our veterinarian _cc781905-5cde-3194-bb3b-136bad5 Treated with pesticide. I follow the owners in a particular way for at least 1 year, until the insertion in the new families is not completed and cmq I will always be available for anything for "the puppy's whole life" and in case something happens, I am available to take it back even in age adult. I like to be and remain a point of reference for new families, of course if this is also your desire. Puppy leaves my house with a kit which includes feeding school, tutorials, pdf videos, food barf. ∆ ∆ ∆ ∆ _d04a07d8-9cd1 -3239-9149-20813d6c673b_ ∆ _cc781905-5cde-3194-bb3b-136bad5cf584bd9-136bad5cf587d_-136bad5cf587d_ 136bad5cf587d_ 136bad5cf587d5 -134bd8 -5398395 -3395 -3c584905- 20813d6c673b_ ∆ ∆ ∆ _cc781905-5cde- 3194-bb3b-136bad5cf58d_ ∆ ∆_cc781905-5cde-3194-bb3b-136b3b-136bad5cf58d_ ∆_cc781905-5cde-3194-bb3b-136b3b- 136bad5cf58d_ 9149-20813d6c673b_ ∆ _cc781905-5cde-3194-bb3b-136bad5cf584a ∆ _d08 -9cd1-3239-9149-20813d6c673b_ ∆ FOR MORE INFORMATION ON THE NORWEGIANS OF THE FORESTS, CHARACTER , FOOD , CARE , TEST , PRECAUZONI , HISTORY AND LEGENDS , PEDIGREE , HOW MUCH DOES A NORWEGIAN PUPPY COST, WE INVITE YOU TO VISIT VARIOUS SECTIONS OF OUR WEBSITE 134

leggende gatti norvegesi delle foreste The True * Origin of the Norwegian Forest Cat Written by Laura Achenza Traveler, whoever you are, you have come here to hear our story, and I ask the wind to let me tell ... *** At the beginning of time Odin, the Exalted, the father of all, created the whole universe and divided it into nine worlds so that every creature would find its place in it; in fact, their forms were many: giants of the mountains and Thursi of the frost, dwarves incomparable master craftsmen, dark elves blacker than pitch and elves of light more beautiful than the sun; Man was entrusted with Midgard, the middle earth, while the luminous gods, the ASI, lived in the stronghold of Asgard and there, from their magnificent thrones, still today observe and rule all the worlds…. Two crows are faithful companions of Odin who every day, at dawn, fly into the worlds and in the evening, at sunset, return, bringing back to their Lord everything that happens there. One day they reported to Odin that the Men of the Northlands were sad and lonely in the long winter night, in which darkness and cold reign supreme. All the ASI were then summoned to the council and each of them proposed to give men their own prodigy: Odin his horse Sleipnir who knows no fatigue and rides in the air, on land and on water; Thor his precious hammer to break the ice; Sif its long and luminous hair to dispel the darkness; Freyr his magical vessel, which always has favorable winds as soon as the sail is raised, and .... Freyja offered her cats .... The ASI Assembly established that, although all of them were prodigious gifts, the one that could truly warm both the body and the heart of the northerners were ... the cats of Freyja. _cc781905-5cde-3194-bb3b-136bad5cf58d _ *** Freyja will therefore have to part with the beloved company of her cats, but she, the goddess of love, knows that there is no better way to spread it around the world! He then decides, for the greater comfort of men and his own cats, to give them an additional magical banner: the sound of Love and Peace that will resound, in Midgard, every time a cat decides to purr .... and Freyja, every time, will be there to listen to him ... In return, however, he asks that his cats spread to every corner of the world, so that everyone can enjoy their magical presence. Therefore the ASI decide that Freyr's vessel will also be given as a gift so that the men of the North can sail everywhere and bring, in honor of the generous gods, Freyja's cats throughout Midgard . _cc781905-5cde-3194-bb3b-136bad5cf58d _ *** At the winter solstice, in the darkest night of the year, the vessel was laid down in a long fjord sheltered from the winds, which will therefore take the name of Sognefjord, the fjord of the dream, and Freyja accompanied her cats to the edge of the forest which stood near a village on a clearing; from that day, his cats belonged to the Norwegian forests and the world of men. Men honored the ancient pact with the Gods, and for centuries they carried the mighty creatures on their ships to every known or hitherto unknown land ... Other Men came and, renewing the covenant, they wanted to give recognition to the race which they called, in memory of their first home on earth, Cats of the Norwegian forests. In their homage they still bear these names proudly today: Vikjngs, Disir Nornir, Freya's Chariot, Of the Ancient Runes, Yggdrassil, Aesir, Lifthasir, Drakkar, ASI e Vanir, Asgard, Vanaheim, Niflheim, Alfheim, Svartalfheim, Midgotheim, Midgardheim Odino, Thorr, Freyja, Bestla, Vidharr, Verdandi, Vali, Var. _cc781905-5cde-3194-bb3b-136bad5cf58d _ *** Now my story has come to an end, but I want to reveal to you one last arcane secret: when your Norwegian cat stops enraptured, and his gaze seems to be playing with the air, know that he is in front of Freyja and for a moment, once again, together, they wander through the nine worlds… ..and you too, if your bond with him is intense, you will be able to see them in the distance. Therefore, remember to love and honor Freyja's gift forever ... Note I drew inspiration from historical texts and literary sources of Norse mythology, but I would like to clarify that the story is pure fantasy, and wants to celebrate and cloak these cats that I love immensely with noble origins. It is therefore a tribute to them and to all fans of this splendid breed.

colorazione dei gatti norvegesi, silver, smoke, shaded, causati da un gene che scolora il pelo partendo dalla base, colorandolo dei toni dell argento. ( esistono 2 teorie) SILVER AND SMOKE "Il gene that makes the Norwegians shine" In some Norwegian cats the brindle design contrasts, instead of on the usual yellowish background on a background white / bright silver; in others, when they walk, the hair opening reveals the sparkle of an icy fog. These cats are always a sight e depending on the design of the coat called_bsc7905-bbc3b3-81d81-3194b9905-bbc3cc3-3194bd81-136b905-3194bd81-136bcc3 -136bad5cf58d_ (smoke), _ cc781905-5cde-3194-bb3b-136bad5cf58d_ Silver (silver), _ cc781905-5cde-3194-bb3b-136bad5cf58d7_Shaded5-136bad5cf58d_Shaded5-136bad5cf58d_Shaded5 Whatever name they are given, they all have one feature in common: the presence of a gene that makes the pigment at the base of the hair disappear completely, the area closest to the skin of cats where the hair is already normally less densely colored due to the effect of body heat on the melanin granules. The gene in question is dominant, and is indicated with the letter "I" capital Smoke, Silver and Shaded obviously can exist on all colors of Norwegians. Below you will see a large gallery of images. SMOKE: Cats are called "Smoke" in which the "I" gene, which discolours the hair at the base, is present on a "NOT TIGERED" cat The root of the hair (from 1/3 to half of the length) is silver white, while the rest is colored: the stationary cat seems to be solid in color, only when it moves can you see the contrast at the base of the hair. Often one can notice a gray shade of the black fur which forms a rich silver colored ruff. Smoke puppies often have a brindle pattern (phantom brindle), which usually disappears within the first year of life. SILVER Cats are called Silver in which the "I" gene that discolors the hair at the base is present on a "TIGRATO" cat The tab design which often includes darker and lighter bands, usually yellowish, in the presence of the silver gene, _cc781905-5cde-3194-bbcfb-136d of a very bright ice white, on which the design will stand out in a particular way. Often on puppies, up to the age of 5/6 weeks is not easily recognizable. Photo gallery of SMOKE cats with and without white Photo gallery of SILVER cats with and without white Hints of Genetics Feline genetics: a combinatorial approach Massimo Picardello University of Rome “Tor Vergata” Department of Mathematics 00133 Rome, Ital y The silver colors (smoke, silver). We now turn to the silver-colored varieties: smoke, silver tabby, shaded silver and chinchilla (and their pheomelanistic equivalents, sometimes called cameo). In these varieties, the base of the hair is depigmented, silver white, without traces of reddish tones. In some silver tabbies, however, the color reaches the root of the hair in the brindle areas. Silver tabbies are brindle cats whose agouti areas are a livid silver hue, with high contrast to brindle. Chinchillas have no traces of branding (in the case of an ideal specimen), and only the tips of the hair are colored (tipping). In shaded, the color on the tips of the hair covers about one third of the length (often less). In the smoke, the silver part is between a third and the initial half of the hair, the rest is colored. The one-gene theory In the past, a single main gene, , was considered responsible for silver colors. indicated with I (inhibitor), responsible for the braking action on the production of pigment and also for the elimination of the reddish shades typical of eumelanistic colors, especially in the agouti areas of brown tabbies. The mechanism by which a single gene can produce such different color varieties is as follows. Smokes are non-agouti cats, and the I gene lightens and bruises the base of the coat, giving it a pure silver color, evenly over the whole body. The braking action of the pigmentation produced by this gene can be limited to about a third of the hair, or be greater, reaching the middle of the hair or slightly beyond. All other silver varieties are agouti. Chinchillas and shaded are agouti with a ticked pattern. (Important warning: Chinchilla or shaded silver puppies often display a mackerel or classic [= blotched] phantom pattern, which disappears as they grow. This does not mean that these puppies are not genetically ticked. Ghost bands are common in ticked puppies: ad for example, they are sometimes seen in the Abyssinians. At most, the presence of phantom stripes may be an indication that the cub is heterozygous for the ticked pattern, but this is not necessarily true). Gene I lightens the base of the hair, but without its effect the hair would not have been all pigmented: there would have been various agouti bands, lighter. The action of I brightens the brown tones of these bands and transforms them into silver as well. Since the lightening due to the inhibition at the base combines with the further silvering of the agouti bands above the cut level of the inhibitory action, the percentage of silver hair extends, and reaches about two thirds in the case that the gene I operate in a mild way (shaded silver), and on all the hair except the tip (the last band of color of the ticked hair) in case the action of I is accentuated (chinchilla). That the chinchilla and shaded silver of eumelanistic colors are agouti is also clear from the fact that the skin of their nose is brick red (or deep pink) with the typical external border of brindle. Finally, the silver tabbies are brindle with a classic (blotched) or mackerel / spotted pattern. it is clear that the action of gene I is much more visible on the agouti areas than on the markings of the brindle, and this gives rise to the brindle design with very high contrast. But just by examining the silver tabbies we see that the genetic explanation of silver colors cannot be based on a single gene. In fact, if this were the case, the brindle markings should be lightened at the base, as in the smoke. But if on the one hand we see silver tabbies of this type, on the other hand we see many others in which the color reaches the root in the brindle areas. Geneticist Roy Robinson, in his well-known book Genetics for Cat Breeders (Cambridge, 1972, 2nd edition), suggests that on brindle areas the action of color filling overpowers silver lightening. But this seems strange, given that on smoke, where the same action occurs on the whole body, the opposite occurs. In any case, it would not be possible to explain why in some silver tabbies there is basic lightening in the brindle areas and in others not. The two-gene theory The most modern theories - see the articles by J. Jerome, TICA Trend vol. 13 n. 6 (Dec. 1992 / Jan. 1993) pg. 14 and TICA Yearbook 12 (1991), pg. 218- assert that two distinct genes act on silver, one responsible for the inhibition of the pigmentation at the base of the hair, the other for the elimination of reddish tones (bleaching, bleaching). We will call this second gene the "silver gene" (Sv). The inhibition gene, on the other hand, we will indicate with I, warning the reader on the fact that some recent texts denote with Sh the mild form of the inhibition gene, responsible for the shaded mantle, and with Ch the intense form, responsible for the chinchilla mantle . But there is insufficient evidence that the difference between the two effects is due to two alleles of the inhibiting gene rather than the action of a group of supporting polygens, and indeed the transition from shaded to chinchilla mantle is quite gradual. than net. Therefore we will not refer here to the alleles Sh and Ch, confident that the reader can now easily adapt the conclusions to the context of articles or books that instead refer to them. Therefore, in these notes we do not distinguish genotypically shaded silvers and chinchilla, attributing the difference to secondary polygens. At this point, the correspondence between the color varieties seen above and their genotypes is clear: Smoke: aa I- Sv - Shaded silver and chinchilla: A- TaTa I- Sv - Silver tabby mackerel / spotted: A- TT I- Sv - if the root of the hair in the brindle areas is silver (silver tabby shell or shaded, depending on whether the silvering is accentuated or reduced); branding is not silver. Silver tabby classic (blotched): A- TT ii Sv - if the hair root in the areas of A- tbtb I- Sv - if the root of the hair in the brindle areas is silver (silver tabby shell or shaded, depending on whether the silvering is accentuated or reduced); branding is not silver. A- tbtb ii Sv - if the hair root in the areas of We have indicated the ticked design gene in shaded and chinchilla as homozygous because the dominance of the design genes is only partial. For example, the TaT combination produces a hybrid design, with diffuse ticking partially superimposed on a mackerel design (mainly on the legs and tail, but often a little also on the flanks). If this is the genotype, the uniformity of the tipping is lost. Of course, cats with these characteristics would still be registered as shaded or chinchilla, but their color would be less consistent with the standard. It should be noted that an ideal silver tabby does not need the action of the inhibition gene I to achieve a perfect contrast between base areas and stripes. In fact, the silver Sv gene lightens and transforms the agouti bands of the hair in the base areas into a pure silver color, which therefore results silvery and livid. Furthermore, the absence of the shading gene I causes the brindle areas to be intensely colored up to the root, increasing the contrast, especially on black silver tabbies, where the elimination of reddish tones makes the black color more intense. is alive. Other color varieties predicted by the two-gene theory; the golden If we substituted genotype I- with genotype ii in shaded and chinchilla, we would obtain A- TaTa ii Sv-. Phenotypically, these cats should be silver ticked tabby in the variant ii, not shaded, that is, with a black band at the beginning of the hair, in contact with the skin. However, it does not appear that these cats were ever produced (see below for further comments). Similarly, if you do the same in the smoke, you get aa ii Sv-, which phenotypically corresponds to solid cats but without traces of rufousing, that is, reddish shades of the hair in eumelanistic colors, or warm tones in pheomelanistic colors. These cats can be obtained by crossing silvers heterozygous for the I gene. If cats of this type are then further crossed with each other, kittens of genotype ii sv sv can be born. From a line of smokes (non-agouti), normal solids are obtained in this case; from a line of shadeds or chinchillas, let's say homozygous agouti AA TaTa, normal ticked tabbies are obtained (like the Abyssinians; however all the shaded and chinchillas are selected against rufousing, that is in favor of polygens of redness of the hair that favor the more bruised, and therefore cannot be expected to obtain, from shaded parents or heterozygous chinchilla Ii Sv sv, ticked tabby puppies with warm reddish tones of the Abyssinians). But if instead shaded or chinchilla heterozygous for Sv and the inhibitor gene I are crossed, it will be possible to obtain puppies with the I-sv sv genotype. These puppies will be ticked tabby with the base of the hair lighter, but without the bruises produced by the sv gene (but still with not too bright reddish tones, due to the polygenic selection against rufousing). The base shade will be gold instead of red. This variety of color, in the eumelanistic versions, is called shaded golden. The corresponding pheomelanistic varieties are too close in phenotype to red (or cream) ticked tabbies to be classified as a variety in their own right. Similarly, golden tabbies can be obtained by crossing silver tabbies heterozygous for the Sv gene. Here are the genotypes of golden, compared to those of brown tabbies: Shaded golden and chinchilla golden (golden shell): A- TaTa I- sv sv Golden tabby mackerel / spotted: A- TT I- sv sv (note that the root of the hair in the brindle areas is depigmented: golden tabby shell or shaded, depending on whether the depigmentation is accentuated or reduced); Brown mackerel / spotted tabby: A- TT ii sv sv (the root of the hair in the brindle areas is not depigmented.); Golden classic (blotched) tabby: A-tbtb I-sv sv (note that the root of the hair in the brindle areas is depigmented: golden tabby shell or shaded, depending on whether the depigmentation is accentuated or reduced); Brown classic (blotched) tabby: A- tbtb ii sv sv (the root of the hair in the brindle areas is not depigmented.). Unverified predictions of the two-gene theory The reader must be warned that this genetic pattern is not entirely satisfactory. In fact, from it we deduce the existence of phenotypes not observed so far. For example, the aa I-sv sv genotype should correspond to a "golden smoke", ie a smoke with a base color of gold rather than silver. But to date, a cat with the color of golden but non-agouti has not been produced. Therefore, it may be assumed that the sv gene is inactivated on non-agouti cats. In this case, the genotypic difference between solid eumelanistic cats with or without traces of rufousing would be due only to the action of polygens. As already observed, also for the silvers (not golden) there is a dubious phenotype: the one corresponding to the genotype A- TaTa ii Sv-. It should be silver cats but not shaded, that is without depigmentation at the base, and with ticked design. As already observed, under the action of gene I the agouti bands become silver, and for example it is possible to see the effect of multiple alternating silver and black bands in the agouti areas of the black silver tabbies mackerel or blotched not depigmented at the base. But in case the design is ticked, we should have a phenotype with all the fur with alternating silver and black bands, and black at the base (in case the base color is black, of course). It is not known that cats of this type were ever produced. It can perhaps be assumed that the Sv gene is active only if gene I is active, and in fact it modifies and reinforces its action. The two additional assumptions we have made in this section in order to "rescue" the two-gene theory are equivalent to assuming that the entire Sv / sv allelic series is inactive except if gene I is present. No research has been done yet. experimental enough to confirm this conjecture. The theory of the golden factor If this last conjecture is true, in fact the two-gene theory becomes equivalent to another genetic model, which was introduced before the theory based on two independent genes. In this previous model, golden were explained by introducing, however, an additional gene g, whose epistatic action causes golden color instead of silver at the base of the silver coat (as is done to explain the Maltese dilution starting from gene B, with the addition of an epistatic gene d). The dominant allele G has no effect (does not cause a golden color at the base). Finally, it should be noted that the Sv gene causes a lack of rufousing in eumelanistic colors, but much less in pheomelanistic ones. There are red smokes and red shaded silvers whose tipping is quite hot red (especially in smokes). The golden theory = brown ticked tabby Another genetic scheme that was considered consisted in identifying the golden ones with simple cats of ticked tabby color (whose design is ticked all over, without any stripe of brindle; in TICA it is called "agouti tabby"). In fact, because the color of the coat is sensitive to temperature, it is almost always true that ticked tabby cats have a lighter coat base (open the coat of an Abyssinian!), And often the golden and ticked tabby phenotypes are difficult. to distinguish. Faced with the difficulties indicated above, deriving from the two-gene model of silver colors, this simple conjecture that golden is nothing other than ticked tabby is suggestive. In fact, the only phenotypic trait that this theory does not explain is the fact that some ticked tabbies are not very clear at the base while others (the golden ones) are very clear, but perhaps the difference could be attributed to the action of a group. of modifier polygens. Despite the difficulties, however, we will continue the discussion based on the two-gene theory, because it gives the most satisfactory explanation for the existence of two types of silver tabbies: those with black tabby areas up to the root and those with depigmented tabby areas. at the base of the hair. However, we observe that, as regards the most important phenotypic traits, this theory that identifies golden with ticked tabbies provides the same percentages of probability for crosses as the two-gene theory. The Broadband Gene Theory Finally, alternative theories have recently been considered in which two genes appear, but with different effects from what has been said above (contributions by H. Lorrimer on the Internet Fancier's List, March-April 1995). In fact, one gene is still the silvering (rufousing elimination) silver gene, but the other would be a gene that causes broadening of the light bands in agouti hair, called wide-band (Wb). However, it should be noted that, due to the large degree of variability in the length of the tipping, it would be more appropriate to speak of a group of broadband branding polygens. In this theory, the problem related to the fact that golden are only agouti (golden smokes are not observed) is obviously solved, but on the other hand the smokes are not explained, which are not only silver (i.e. without rufousing), but also lightened at the base. As these are non-agouti cats, this aspect of their phenotype cannot be due to genes that widen the clear agouti bands. On the other hand, a variant of this theory can be used to justify a theory advanced in some books: that chinchillas and shaded silver are not necessarily of ticked design. The ticked design, together with the silver inhibiting action, ensures that the color is limited to the tip of the hair (tipping). For g | the other designs this would not be the case, and furthermore the length of the tipping would be far from uniform. But if the agouti bands were dilated by a group of wide-band polygens, and then silvered by the silver gene, then the tipping would be confined to the tip of the hair, and there would be an acceptable degree of uniformity. The theory [golden = brown ticked tabby + broadband polygens]: finally an exhaustive genetic model for smoke, silver and golden! Let us return to the theory that identifies the shaded golden and ticked tabby color varieties. This is 19 the most satisfactory theory we have encountered, except for the crucial circumstance which does not explain the partial depigmentation at the base of the hair, a fundamental characteristic of the shaded golden phenotype. On the other hand, there are many cats registered as golden and with varying degrees of depigmentation, some even with almost no depigmentation ... Therefore it must be assumed that shaded golden depigmentation is not the effect of a main gene with net action. (or depigmentation or full color), but the result of the gradual (and sometimes very reduced) action of a set of polygens. But there is already a candidate for this group of polygens: the broadband modifiers introduced in the previous section. Then shaded golds would be nothing more than ticked tabby with broadband base brightening (aside from eye color, which we'll talk about later). If so, everything is fine: the golden must be tabby (no golden smokes!) And partially depigmented at the base (but the amount of depigmentation varies from one specimen to another depending on how much the effects of the individual modifier genes are added together. broadband). Naturally, in this theory the ideal shaded golden results result from crosses of shaded silver heterozygous for the silver and homozygous for the ticked pattern. What if the shaded silver parents are heterozygous also for the ticked drawing? For example, parents might carry the mackerel factor. In this case, if instead of being silver they were brown tabby (no silver), the parents would have brindle lines on the legs, chest and tail, due to the partial dominance of the ticked allele on the mackerel. But as they are shaded ilver, the branding lines will be there, but they will be faint. And in fact, there are some shaded silvers with faint traces of branding (especially from puppies, before the agouti gene reaches maximum effect). However, their golden kittens are not silver, they are brown ticked tabby, and if they carry the mackerel allele the bruises will be clearly visible on the paws, a very undesirable feature in shaded golden, but frequent. To complete the picture, the broadband modifiers transform the other designs (spotted, mackerel and classic) into the corresponding golden varieties (golden spotted tabby, golden mackerel tabby and golden classic tabby respectively), in which the agouti areas are partially depigmented at the base and have warm apricot tones. Now we finally have a satisfying golden theory. However, in order to be compatible with the silver one, it is necessary to review and correct the latter. We can no longer hypothesize two genes for silver, one for depigmentation and the other for silvering. Instead we have to go back to the one-gene theory, an inhibitory gene that we will continue to call I and that simultaneously causes silvering and depigmentation, as in smokes, for example. It should be noted that the golden genotype is not due to gene I (indeed the golden ones are heterozygous ii): the depigmentation at the base of the golden hair is instead due to the elongation of the first agouti band due to the action of the broadband modifier genes. We do not introduce a distinct main gene that causes depigmentation, otherwise, as we have seen, the theory would predict the non-existent golden smokes (golden non agouti, depigmented at the base of the hair). On the other hand, it would seem at first glance that this loses the brilliant explanation that the two-gene theory gave regarding the fact that some silver tabbies are depigmented at the root of the hair in the brindle areas and others are not. But this genetic trait can be explained in some other way. The gradual level of depigmentation at the base of the hair in the brindle areas may be caused by modifier genes similar (or possibly identical) to broadband modifiers, whose action is limited to tabby cats. With a slight conceptual abuse we can imagine that these additional modifiers belong to the same group as the broadband polygens (in reality this is not the case, because the latter act on the agouti base areas while the former act on the brindle areas: but in reality they meet many golden in which broadband depigmentation also occurs on the areas of tabby marks ...). In summary, compared to the two-gene theory our new genetic model retains the inhibitor gene I (which is now considered responsible for both depigmentation and silvering, as in the one-gene theory), and replaces the second gene with a group of polygens. , which we will denote by Wb (for "wide-band"). What is particularly satisfying is the fact that the gradual action of broadband modifiers can explain the difference between the shaded silver and chinchilla phenotypes. Unfortunately, however, we are assuming that these are polygens, which therefore are not subject to the simple and direct rules of Mendelian genetics for the main genes. In order to analyze the mathematics of this new model it is necessary to resort to statistics, the results of which are too fine to be clearly distinguished in the phenotypes. Therefore it is not easy to present the results by means of cross tables. Silver and golden It should be noted that the deepening of the single gene theory presented in the previous section, as well as the variant introduced previously, excludes the possibility of cats at the same time silver and golden: the silver phenotype is a consequence of the inhibitory gene, which the golden they do not have. Both shaded silver and golden have broadband type color suppression or lightening genes, or polygenic modifiers of the same type. In the latest version of the theory that we presented immediately above, the golden pattern is caused by the suppression action of this type of genes superimposed on hairs with already banded coloring due to the Agouti gene: thus, if the tabby design is the ticked, you get the perfect golden pattern, uniform throughout, but if the design is mackerel, spotted or (even worse) blotched, then more or less large areas of tabby marks become evident with full color, not very lightened, only orange in color in the band at the base. These patterns could be classified as golden tabby (mackerel, spotted or blotched, respectively). Since in such cases the areas where the coat has a broad golden base are less broad, these phenotypes are more difficult to determine with certainty, particularly in kittens, especially if they are phantom patterns in young cats due to heterozygous tabby genotypes with a only ticked allele, and some origins books (for example that of TICA) do not recognize them. Finally, it should be noted that the action of broadband suppression genes creates long agouti bands of pheomelanistic color, which are difficult, if not impossible, to recognize on cats of already pheomelanistic base color. If the lightening band is not long this is easier, just like in the case of the tortie ticked tabby pattern. Eye color of shaded and golden: an example of persistence? In relation to the similarity between golden and ticked tabbies, it should be remembered that in ticked tabbies the color of the eyes is gold or copper (in the case of Persians) or also hazel or yellow or yellow-green or green (in the Abyssinians). In the shaded silver or chinchilla of eumelanistic colors the eye color is not gold or copper, but emerald or blue-green. Instead, in the shaded silver or chinchilla of pheomelanistic colors, the eye color is copper. With appropriate crossings and consequent selection, it has been possible to introduce the gold or copper color in the eumelanistic shaded silver: the corresponding standard, recognized by some feline associations but not all, is called black shaded (in England, pewter). So the difference between an eumelanistic golden and the corresponding ticked tabby, although it may be barely visible in the color of the hair, is still evident in the color of the eyes, emerald in the first but not in the second. In silver tabbies, on the other hand, the eyes are green or yellow-green or gold, with a preference for green. In the shaded and in the pheomelanistic silver tabbies the color of the eyes is copper, and in the corresponding tortoiseshell varieties it can be copper or emerald (shaded) or green (silver tabby), with preference for copper. The existence of black shaded with gold eyes reveals that the emerald color of the eyes is not caused by the silver gene. It is interesting to observe that, although the color of the eyes, emerald or copper, is largely determined by a main gene, it is not common to meet eumelanistic shaded silvers with copper eyes: the emerald color occurs more frequently. So, although the color of the eyes and the hair depend on different genes, somehow there is a persistent link between the shaded silver coat and the emerald color of the eyes. This link is perhaps due to the fact that the main genes that cause this eye coloring are located on the same chromosome as the genes that cause the shaded silver color of the coat, and therefore the genetic transmission of one and the other occurs together. A situation of this type, which is called persistence, modifies the probabilities of concomitant genetic transmission of the two genes, which are very different from what they would be for two independent genes. To break the persistence, in fact, it is necessary that in the close contact of the two homologous chromosomes during the pairing that precedes meiosis, a genetic recombination takes place, that is a molecular exchange between the two chromosomes. The action of the I and Sv genes is epistatic on that of the solid color, eumelanistic and pheomelanistic genes, but not on that of the white (W) and piebald spot (S) genes. Obviously, the W- B- A- TaTa I- Sv- genotype corresponds to a phenotype in which white masks the shaded silver color on the coat, but the eye color can be either blue (due to the depigmentation action that the gene W can exert on eye color) or emerald.

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Falsi miti sui gatti norvegesi , come : "le femmine non hanno la gorgera", le femmine sono meno coccolone, e tante altre false credenze..... False Myths and Curiosities About Norwegian Cats Wrong news, hoaxes, false myths, wrong beliefs, curiosities and everything else that always has you said about cats, which does not correspond to the truth. The page is always evolving, with patience I will add below everything that in my 14 years of breeding I have heard about Norwegian cats, but which is absolutely WRONG! If you want to contribute to this page by giving us some falsa news or incorrect information, you can write to me at: infogattinorvegesi@gmail.com_cc781905__5cde-319bad2 3194-bb3b-136bad5cf58d_ Come back and visit us. False Myths 1.0 🤣🤣🤣🤣 " Female Norwegian Forest Cat DOES NOT have a Gorgera (or also called a mane)" 🤣🤣🤣🤣 These in the picture are all my females _cc781905-5cde-3194-bb3b-136bad5cf58d _....... so as you can see this is wrong news; _cc78190- 5cde-3194-bb3b-136bad5cf58d_the female Norwegian forest cats have of beautiful, thick and majestic gorges. Whether or not to have the Gorgera "does not depend on the sex of the cat, but depends on the age, _cc781905-5cde-3194-bb3b-136bad5c5cds -5cde-3194-bb3b-136bad5cf58d_ especially if born in summer, they will have very little undercoat e for this reason they could look like short-haired kittens, but ......._ cc781905-5cde-3194-bb3b -136bad5cf58d_do not be fooled! At the end of the second moult, about the second year of age, (some even earlier) when they begin to be adults, they will put their thick "gorgera" (the Norwegian forest is subject to the total loss of the undercoat in spring, therefore only what is called "guard hair" more bristly and longer, water-repellent will remain.)

La salute dei Gatti Norvegesi non viene lasciata al caso...Siamo seguiti da Veterinari specializzati... Our Veterinarians, Specialists and analysis laboratories -All of our Cats are tested annually and occasionally for genetic diseases and for: ANNUALLY -HCM- Hypertrophic Cardiomyopathy: read more OCCASIONALLY-HD- Hip dysplasia: learn more OCCASIONALLY-Fiv OCCASIONALLY - Felv GENETICO- GSD 4: learn more 1 VOLTA -PKD - Polycystic Kidney Syndrome: find out more GENETIC -PK-Def-Pyruvate Kinase Deficiency: read more : TO THE INSERTION OF A NEW CAT -TriTrichomonas - Giardia-5c3b-136bad5cf58d_bad5-136c3 -5cf58d_Giardia-136 bb3b-136bad5cf58d_Felv EACH LITTER- Giardia EACH LITTER- Parasitological faeces All NEGATIVE in 2019 IN ADDITION: All our puppies leave our home with their health record which includes: Veterinary certificate of good health. Parental tests (see above) e when possible also those carried out on grandparents, especially with regard to hypertrophic cadiomyopathy (HCM) Pyruvate Kinase Deficiency (PK-Def) Pyruvate kinase deficiency (PK-Def for short) is a genetically based disease. Pyruvate kinase is an enzyme found inside red blood cells and controls their ability to produce energy to survive. When the enzyme is deficient, the red blood cells undergo premature destruction (erythrolysis) and this therefore leads to a reduction in their blood number. The resulting anemia is therefore the primary consequence of the deficit. However, the diagnosis of PK-Def on a symptomatic basis can be very difficult: since the cat's body is able to adapt the production of red blood cells in the face of their decrease, anemia can occur occasionally or intermittently. The development of the disease is slow, in most cases, with vague symptoms such as lethargy and loss of appetite. However, there is the possibility that the cat will develop very severe anemia, which could put its survival at risk. PK-Def is a congenital disease with a genetic basis, for which there is no cure and, since anemia is very often mild, the clinical signs may not be immediately obvious and therefore the condition may be underestimated for a long time. There are cases of deaths within two years, but also cases where the life expectancy is long (over 8 years). It is essential for a cat affected by the disease that its living conditions are optimal and that stressful events are avoided as much as possible. The disease is typical of the Abyssinian and Somali breeds, but can be found in breeds that have undergone hybridization with the Abyssinians or Somalis as well as in domestic cats. Genetic profile PK-Def is an autosomal recessive disease. This means that a cat can be defined: normal: does not have any copy of the defective gene, will not become ill with the disease or pass the disease to the offspring carrier: in the pair of alleles that define the disease, there is a defective allele. The cat will never get sick, but it will be able to pass the mutation to the offspring affected: the pair of alleles is composed of two defective chromosomes. The cat will develop the disease and will certainly pass the mutation on to the offspring. In case of mating between two PK-Def carriers, since both parents have a copy of the defective gene, they will be able to pass the mutation to the offspring. Specifically, two carriers will generate 25% of puppies affected by the disease, 50% of carrier puppies and 25% of healthy puppies. In case of mating between a carrier cat and a healthy cat (ie not a carrier of the deficiency), we will have 50% carrier cats and 50% healthy cats. In a mating between two affected cats, 100% of the offspring will be affected. Management on the farm From a breeding point of view, affected cats should not be used for breeding. It is possible to use carrier cats, obviously defining a control and negativization plan of the line, to manage the pathology as much as possible and go towards the eradication of the pathology in the breed. In the Abyssinian and Somali breeding, due to the small number of subjects currently working with, the breeding plans include the conscious use of carriers, because their carpet sterilization would involve serious problems of numerical consistency of the gene pool. In general, what needs to be done when deciding to use a carrier on the farm is to mate it with non-carrier cats, test the litter and prefer to continue working on the line keeping mutation-negative cats on the farm. In this way, the dual objective of "cleaning" the bloodline and, at the same time, of preserving it will be achieved. The UC Davis study As we have already published in the previous article, News on PK-Def in Norwegian Forest Norwegian, un recent study by UC Davis_cc781905-5cde-3194_badica-136b3b the disease is also present in the Norwegian Forest Cat, although at present we do not yet have precise indications on the gene frequency and on the consequent percentages of carriers and affections in the breed. We therefore suggest, in this first phase, to test your cats and to submit the results of the tests to the public databases for collecting the results, to allow a more in-depth study on the spread of the mutation. Where to take the test Since the mutation found in the Norwegian Forest Cat is the same as the one found in the Abyssinians and Somalis for which genetic testing already exists, it is possible to test your cats in different laboratories that are already certified to perform the test. It is possible to perform the test with either a blood sample or a buccal sample. Here is a non-exhaustive list of the laboratories to which you can turn: Genefast (Italy - agreement with Anfi) Vetogene (Italy) UC Davis (USA) Laboklin (Germany) Text taken da http: //www.clubnorvegesi.com/ Type IV Glycogenosis GSD IV (glycogen storage disease type IV) is part of a group of diseases, called glycogenoses. Type IV affects the Norwegian Forest Cat. They are well described pathologies also in humans, classified as rare diseases. In a normal organism, the excess glucose obtained from the diet or from the conversion of proteins and fats is stored in a branched chain of polymers, glycogen, built using the enzyme GBE (glycogen branching enzyme) as a catalyst. When the body needs energy, glucose molecules are removed from the glycogen and released into the bloodstream or used by the tissues. The ability to efficiently add and remove glucose from glycogen depends on the complexity of its branching structure. GSD IV is a hereditary deficiency of the GBE enzyme: affected subjects store an abnormal form of glycogen which, therefore, leads to an insufficient use of glucose. Hence GSD IV can be considered a chronic hypoglycemia that inexorably worsens towards death (because the body is unable to use glucose effectively). In cats, affected kittens die a few hours or days after birth, most likely because they do not have enough glucose to survive delivery and the first hours / days of life. The late form is rarer, for which the puppy is healthy up to 5/7 months and then suddenly shows a stunted growth and widespread weakness, with the following symptoms: high hyperthermia (over 40 °), insensitive to corticosteroids intermittent and generalized tremors that become permanent intermittent, "hiccup" weakness muscle weakness, followed by muscle atrophy, fibrotic contractures of joints that lead to movement and feeding difficulties and that require continuous assistance from the owner quadriplegia Affected cats can survive up to 10/14 months. They die of cardiac arrest, sometimes after coma. The disease is fatal and there is no cure. It is possible that it is confused with neonatal isoerythrolysis because the symptoms can suggest both pathologies. The only thing that can be done is the test (genetic for GSD IV, blood group identification with B allele typing for neonatal isoerythrolysis). Genetic profile The GDS IV mutation is recessive, which implies that the possibility of it expressing itself in the offspring exists only if both parents are carriers of the mutation. There are 3 different possibilities: both parents are healthy (they are homozygous for the normal allele): the children are healthy and will not carry the mutation. if one of the parents is carrier (heterozygous) we will have about 50% carrier and 50% healthy cats among their offspring. When two carriers are mated we get 25% sick cats, 50% carriers and 25% healthy cats. Recall that a carrier cat is a cat that does not have the disease and that will never develop it. However, he will be able to to transmit the mutation to his children. The test A genetic test, on blood or buccal swab, is available to identify not only affected cats but also carriers. Some laboratories that perform this test are: Vetogenic Genefast Antagen Genindexe Laboklin PennGen Each laboratory provides for the sending home, upon request, of the test material (swabs or blood collection tubes), together with the necessary documentation. In some cases it is required, for a correct identification of the cat, that the microchip is applied. Insights FYFE et al. (2007) A complex rearrangement in GBE1 causes both perinatal hypoglycaemic collapse and late-juvenile-onset neuromuscular degeneration in glycogen storage disease type IV of Norwegian forest cats, Mol Genet Metab. 90, 4, 383-92. http://www.pubmedcentral.nih.gov/articlerender.fcgi?tool=pubmed&pubmedid=17257876 Glycogenoses on Wikipedia FYFE et al. (1992) Glycogen storage disease type IV: inherited deficiency of branching enzyme activity in cats., Pediatr Res. 32, 6, 719-25.FYFE (1995) Glycogen storage disease in cats, J Am Vet Med Assoc. 206, 3, 286. GASCHEN et al. In Congenital feline myopathies, Glycogen Storage disease type IV, 360-361 http://www.winterfyre.com/testing/ http://gsd4.de.vu/ v Text taken da http: //www.clubnorvegesi.com/ Anchor 4 Anchor 5 The health of our cats

gatti norvegesi femmine, fotografie gatte norvegesi, peso gatte norvgesi, colori gatti norvegesi, trecolori, tortie, blue, grige FEMALES - FEMALES IT * DISIR NORNIR ERZA SCARLETT Born 07/11/2019 Mother: SE * ZIMEXIS CAPTAIN MARVEL Father: NO * TINGOSKATTENS JARL HCM Negative 2021- GSD 4: negative - PKD negative - PKDef- Negative * PEDIGREE * DE * Afjord's Kamiko Born 11/04/2018 Inbreeding begins in 7th generation = 0.153% Complete inbreeding 4.59% Polaris 15.2% * PEDIGREE * First HCM Screening Sept. 2021 NEGATIVE by Dr. Robert Ghinelli, approved by the Italian HCM Observatory GSD 4: negative PKD: negative PKDef: Negative Info on ticked branding, history, photos and more about Kamiko can be seen on the website Info about ticked tabby, the story, pictures, and more on Kamiko, you can find in her web site Born 8-10-2021 Mother: NO* Maya Alexandra Cornelia Father: NO*Disir Nornir Feanor * PEDIGREE * IT* DISIR NORNIR Karibibite Karin

Maschi del nostro allevamento amatoriale di gatti norvegesi, vivono in casa e nei catrun ( recinti protetti) - Non sono disponibili per monte esterne- PARENTS father: NO * FRU PEDERSENS PEDER STARUP mother: SE * ZYGOT ALLEY-OOP NO * Fru pedersen ISAK SYLVESTER Blood Type A 1st scan HCM -Negative Oct. 2015 2nd scan HCM -Negative Nov. 2017 3rd scan HCM -Negative June 2019 4th scan may 2021 - Negative PKD-PK Def - GSD4: Negative Fiv Felv: Negative 2019 Chlamydia: negative Aug. 2019 HerpesVirus Negative Aug. 2020 CalciVirus Negative Aug. 2020

Come proteggere il gatto da cadute accidantali da balconi, terrazzi. Come costruire una recinzione a prova di gatto, catrun, recinzioni per gatti, foto chiusura How to protect our cats from accidental or voluntary falls _cc781905-5cde-3194-bb58f-136bad5cof windows and balconies Original text, written by Karin Pedrona, the use of all or part of the text is prohibited. I would like you to read these lines carefully and not underestimate. More and more often it happens that purebred cats, especially curious breeds such as the Norwegian, die from stupid domestic accidents, which would have easily could have been avoided. bb3b-136bad5cf58d_ The windows and balconies are a great joy and a source of well-being for our puppies but, at the same time, they are a source of great danger. Above all, purebred puppies are not born from mothers used to getting by, jump from trees, climb, have the perception of emptiness and measures, run away due to a dog, and being exposed to dangers of various kinds. Fortunately and unfortunately the purebred puppies, are born from mothers who for generations have been protected loved and respected by human beings, therefore these mothers are not able to_cc7 -5cde-3194-bb3b-136bad5cf58d_ teach and above all SHOW, what which they can't do and don't do. As you know, mothers are the reference point for puppies and teach them, as human mothers do with their children, everything they need to approach life, with all the appropriate recommendations. (go slowly with that scooter! :-D) it's a pity that no one can teach cats that a fall from the window can be fatal . they will never grow up to be able to learn it, (among other things there are cats with great relapses, precisely because the desire to escape and curiosity is more_cc781905-5cde-3194 -bb3b-136bad5cf58d_strong of danger that they do not perceive due to inexperience) Not infrequently it has happened that a puppy or even adult Norwegian (which like all cats in the soul are great hunters), _ cc781905-5cde-3194-bb3b -136bad5cf58d_ a jump run is enough to catch a fly e so so unaware, _cc781905-5 bbcde-3194_bad fell into the empty window. Over the years, I have had friends in breeders and families who have adopted my cats, who have had very bad experiences ... I often find myself talking about Agra, my mother's cat, which despite being 1.30 meters high di windowsill on which he had great margin not to fall, he wanted to sit right inside a vase of an orchid, which logically with the weight of the cat unbalanced and the cat fell from the 1st floor together with the vase, fortunately breaking only the front legs, the upper lip and the teeth. ... If you don't even have the mitigating factor of being on the first floor ... when the cat or puppy falls, at best it dies .... yes , I said well, because in the second hypothesis ... he remains paralyzed and you will have to squeeze his bladder for life, because they are easily paralyzed. Speaking of the "vasistas", or also called "tilting" windows, we can speak of real death traps :-( When the animal tries to pass into the narrowest corner that forms at the base of the window gets stuck and no longer has the ability to lean on none of the legs, thus remaining in a suspended position that the crushes the nerves and lungs with his every move to wriggle out . A terrible death therefore that can be avoided for example by closing the basement windows with a wire mesh placed outside. In apartments where there are cats, the tilting position of the windows must be avoided. Advice: > You can contact experts in the sector, able to close balconies or windows even difficult problems, these experts in the sector move in various regions of Italy, for example: https://www.facebook.com/reteprotezionegatti.valentino.7?fref=ts > here you can buy here special tilt window grilles > You can buy DIY nets on retiprotezioni , or on Zooplus, . > some photographs included in the album are do-it-yourself, others are of works made by specialized companies, you will see the company logo in the photo itself Original text, written by Karin Pedrona, the whole or partial use of the test is forbidden, to use it write to: infogattinorvegesi@gmail.org Do it yourself solutions Solutions made by professionals leonardo valentino leonardo valentino 13533184_731024593666952_492131190915518 19059201_970665356369540_331344684581157 19145972_970665313036211_867601190900655 13592698_731024633666948_225693550076443 13438814_731024660333612_200265172010061 13592386_731024697000275_338616251574687 leonardo valentino 12743653_671951746240904_867418853715856 ekosistem ekosistem Schermata 2018-10-29 alle 16.44.36 Schermata 2018-10-29 alle 16.44.04 Animalando Ekosystem Italia Animalando Ekosystem Italia Animalando Ekosystem Italia 10291041_252643461606915_4686965870894083723_n.jpg fai da te 10334247_252643831606878_5593615182197474065_n.jpg 10334323_252643584940236_8961473994830189414_n.jpg Animalando Ekosystem Italia Animalando Ekosystem Italia Animalando Ekosystem Italia Animalando Ekosystem Italia Installation of Nets on balconies, what the law says How to secure a garden

gatto maschio silver norvegese delle foreste TINGOSKATTENs JARL _cc781905-5cde -3194- bb5894_ISbad5c3 -owner WORLD'S Cattery -Milano Italy Scan Hypertrophic Cardiomyopathy Issued by Claudio Bussadori Dates 21/11/18 (age 1.4.15) Result Normal - - - RDA (PCR) Date 12/2/2019 Chlamydya- Herpes- Chalices- Mycoplasma Result Negative - - - * Pedigree * Tingoskattens Jarl, M, NFO ns 24, 06/07/17, NO / IT Father: NW SC Sakeena's Aston Martin, JW Mother: Tingoskattens Zona

Progetto per la salvaguardia del mantello con pattern / tigratura Ticked nel gatto norvegese delle foreste. E' arrivata la prima cucciolata Italiana di gattini Ticked ! The Norwegian Forest Cats Ticked Project for the preservation of the TICKED Pattern / Tiger in the Norwegian cat ok but what is the "TICKED"? _ cc781905-5cde-3194-bb3b-136bad5cf58d_ The ticked is a very rare type of brindle in the Norwegian cat, now almost extinct. The Ticked is the classic tigratura of wild animals such as lynxes. After many, many years of research to find an heir to this pattern, finally last year DE * Afjord's Kamiko arrived. If you want to read more follow the link on_cc781905-5cde-3194-bb3b- 136bad5cf58d_ "Project for the protection of the Pattern Ticked in the Norwegian Forest Cat" If you want to contact us you can write to: infogattinorvegesi@gmail.com ... What Peter Clausson Friis called the lynx-cat was perhaps the "real Norwegian Forest Cat." -5cde-3194-bb3b-136bad5cf58d_ there are many similarities; are both large cats, with high legs, round feet, long mane and tufts of hair on the ears. In the Norwegian villages there were many cats of course, but in the ancient legends there is a particular type that is mentioned very often, the Huldrekat ", ...